Pick 9 ND 2012 – Who should we pick? | PUNT ROAD END | Richmond Tigers Forum
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Pick 9 ND 2012 – Who should we pick?

If we take Vlastuin over Mayes I will be left speechelss :-X

Vlastuin is solid, however, too similar to Conca and Ellis. He is a good kick and has nice hands, but lacks pace and endurance, don't need another vanillia hbf. He won't become a midfiedler unless he dramtically improves his fitness. Mayes has better skills, pace and endurance, surely its a no brainer.

Personallly I can't see Grundy getting past Port Adelaide, particularly if the Dogs take Menzel. I would back Port to pick Grundy over Mayes so suggest we give up on that one.
 
Magic will he be available - Mayes that is and its sounds like you agree Vlastuin at #9 is a bit of a reach.

Damn shame we couldnt offload Nahas for a better possie in the first or second round.
 
How does Grundy compare to someone like Patton from last years draft ?
 
Personally, I hope we take Stringer if he lasts to our pick. He could become our Adam Goodes. Massive upgrade in the role Tuck plays, and despite the rumoured immortality of the Tucks, you would think this may be his last season. He could also provide that lead up med forward in the Jack Darling mold, can kick bags, and by all accounts is a competitive beast. If Stringer develops his pre-injury fitness levels, and he seems to be doing that at a sensible rate, he would be an absolute steal at 9, and the answer to quite a few of our deficiencies.
 
craig said:
Magic will he be available - Mayes that is and its sounds like you agree Vlastuin at #9 is a bit of a reach.

Damn shame we couldnt offload Nahas for a better possie in the first or second round.

Not sure. It is shaping as though one of Mayes or Macrae will be avaliable at 9 even with Melbourne picking at 4. Potentially both will be avaliable if Melb has 4 taken off them.

My current top 10 is:

1. Whitfield
2. Toumpas
3. O'Rourke
4. Wines
5. Menzel
6. Macrae or Mayes
7. Grundy
8. Plowman
9. Mayes or Macrae
10. Daniher

The player I really want is Plowman, however, Brisbane are apparently super keen. If the Dogs pick Plowman instead though then there is a chance Grundy may drop to 9, as that could leave Menzel at 7 for Port. I think Brisbane will take Jaksch if Plowman is gone.
 
Baloo said:
How does Grundy compare to someone like Patton from last years draft ?

Chalk and cheese really, Grundy is primarily a ruck who's just learning how to play forward. His tapwork is elite and he's what I consider to be a modern day mobile ruckman. His forward work still has some way to go but the signs are promising and if I was to compare him with anyone, it would be Kurt Tippett in his transitioning days from basketball to football.

Mr Magic said:
If we take Vlastuin over Mayes I will be left speechelss :-X

It would be reminiscent of the Conca versus Heppell decision, both have their strong points. Given we did take Conca last time, I'd be inclined to grab Mayes this around.
 
Coburgtiger said:
I want Troy Menzel.

Not worth the risk reco with a LARS and his brother also done an innocuous ACL i think he did it twice seems that theyve got genetically weak ACLs (same for the browns Mitch and Nathan both done them, Lee Walker ect) yes hes good but id rather a more certain venture pre draft and im sure many recruiters feel the same.

He may go well and star without a hitch ala Judd and Selwood but the concern is there and its percievable.

ACLs are a bit different to shoulders, Stringer too with his leg has no doubt caused much consternation amongst list managers and recruiters.

LARS apparantly aint long term they fray and elongate (stretch) thats why many orthopods dont rate em especially for youngsters.
 
craig said:
Not worth the risk reco with a LARS and his brother also done an innocuous ACL seems that theyve got genetically weak ACLs (same for the browns Mitch and Nathan both done them, Lee Walker ect) yes hes good but id rather a more certain venture pre draft and im sure many recruiters feel the same.

He may go well and star without a hitch but the concern is there and its percievable.

LARS apparantly aint long term they fray and elongate (stretch) thats why many orthopods dont rate em especially for youngsters.

Are you a scientist? Big call on Troy having genetically weak knees.
Also, are you an orthopaedic surgeon? Another big call on the LARS.

For what it's worth, I am actually a scientist, specifically within genetics, and I've had LARS surgery too.

And what you've said is bunk.

Also, neither of Troy's knee injuries were innocuous, both were impact injuries. It's a big stretch to call that a genetic link, and is not backed up by science.

The actual professionals, (i.e. Julian Feller - one of the most respected in the field) have said Troy's knees are fine.

I don't want Geelong to end up with both Menzels, they will make everyone look fools.
 
Hopefully Menzel gets picked up before #9 so we don't have the opportunity to take the risk.

Would prefer a 'safe' option with our first pick and would love one of Wines or Grundy to slide.
 
Coburgtiger said:
I don't want Geelong to end up with both Menzels, they will make everyone look fools.
If the cats get the opportunity they wont be able to call out his name fast enough!
 
Coburgtiger said:
Are you a scientist? Big call on Troy having genetically weak knees.
Also, are you an orthopaedic surgeon? Another big call on the LARS.

For what it's worth, I am actually a scientist, specifically within genetics, and I've had LARS surgery too.

And what you've said is bunk.

Also, neither of Troy's knee injuries were innocuous, both were impact injuries. It's a big stretch to call that a genetic link, and is not backed up by science.

The actual professionals, (i.e. Julian Feller - one of the most respected in the field) have said Troy's knees are fine.

I don't want Geelong to end up with both Menzels, they will make everyone look fools.

For what it's worth, there is absolutely a familial predisposition to ACL ruptures. There are also several genes that code for metalloproteinases that have been associated with an increased risk of ACL ruptures, and other factors such as the anatomy of the tibia and femur, which can potentially be familial, play a role as well.
Daniel Menzels' knee injuries seemed relatively innocuous to me, and I think concern over Troys' knees is warranted, and should be factored in when considering drafting him.

Flynn RK, et al. The familial predisposition toward tearing the anterior cruciate ligament - A case control study. American Journal of Sports Medicine. 2005
 
Does anyone else rate Nick Graham? Is a possibility to be around at our 2nd and 3rd picks.
 
Tigerdog said:
Does anyone else rate Nick Graham? Is a possibility to be around at our 2nd and 3rd picks.

Any relation to Angus ?
 
bandoftigers said:
For what it's worth, there is absolutely a familial predisposition to ACL ruptures. There are also several genes that code for metalloproteinases that have been associated with an increased risk of ACL ruptures, and other factors such as the anatomy of the tibia and femur, which can potentially be familial, play a role as well.
Daniel Menzels' knee injuries seemed relatively innocuous to me, and I think concern over Troys' knees is warranted, and should be factored in when considering drafting him.

Flynn RK, et al. The familial predisposition toward tearing the anterior cruciate ligament - A case control study. American Journal of Sports Medicine. 2005

Thats good postin' BOT. Love the bibliography
 
Both Menzel and Stringer worry the heck out of me, to be using a top ten pick would be taking a huge risk.
 
Best for menzel that we don't recruit him as our luck would ensure that his ACL blows the big one when he's in yellow and black, but will play a long and illustrious AFL career if he gets drafted after our pick.
 
Coburgtiger said:
Are you a scientist? Big call on Troy having genetically weak knees.
Also, are you an orthopaedic surgeon? Another big call on the LARS.

For what it's worth, I am actually a scientist, specifically within genetics, and I've had LARS surgery too.

And what you've said is bunk.

Also, neither of Troy's knee injuries were innocuous, both were impact injuries. It's a big stretch to call that a genetic link, and is not backed up by science.

The actual professionals, (i.e. Julian Feller - one of the most respected in the field) have said Troy's knees are fine.

I don't want Geelong to end up with both Menzels, they will make everyone look fools.

No i am not a scientist.

But as a scientist you would have to agree that the physiologically and biologically the genetic make up and chromasomal link to pre dispositon for injury illness, disease heredation is already founded and proven scientific fact especially including connective tissue disorders,

All tissues in the body are formed of stem cells and dna thus chromosomal defects and genetic variances / malforms predispositions will lead to physiologicacal weakness, malformities, and pre disposition for illnesses or disease, be they auto immune, gentically linked, histological, neurological, haemotalogic, and heredation.

I dont recall Menzell having major trauma to his knees i remember it giving out on him like so many others before and since, innocuous low impact injuries when changing direction and landing jumping etc are what i am on about not having someone crash over your knee Willy T style.

Dodgy Shoulders whilst scary to recruiters are not as scary as major limb fractures and dodgy knees and achilles.

Im betting theres plenty of recruiters chewing their fingernails over this kid and likewise Stringer. For average Joes and veteran athletes they are great, but a number of Orthopods have been less keen as yet to recommend LARS injury dependence of course hence most AFL players still have traditional recos.

There has been studies into heredity and genetic predisposition to connective tissue injuries.

The research has been into into collagen and proteins that make up the connective tissues in partuicular predisposition for Achilles Tendon rupture, Rotator Cuff injuries and dislocations and ACL ruptures, amongst athletes with surprising evidence pointing to increased propensitry amongst female athletes but also identifying a genetic link amongst siblings.





Genetics and Sports

Genetic Risk Factors for Musculoskeletal Soft Tissue Injuries

Collins M, Raleigh SM

Collins M (ed): Genetics and Sports. Med Sport Sci. Basel, Karger, 2009, vol 54, pp 136–149 (DOI: 10.1159/000235701)

Article (PDF 412 KB) Free Preview
Medline Abstract (ID 19696512)


Recommend this chapter

Abstract:
Acute and overuse musculoskeletal soft tissues injuries are common as a result of participating in specific physical or workplace activities. Multiple risk factors, including genetic factors, are implicated in the aetiology of these injuries. Common musculoskeletal soft tissue injuries for which a genetic contribution has been proposed include the Achilles tendon in the heel, the rotator cuff tendons in the shoulder and the cruciate ligaments in the knee. Recent developments in the identification of genetic risk factors for tendon and ligament injuries will be reviewed. Sequence variants within genes that encode for several tendon and/or ligament extracellular matrix proteins have been shown to be associated with specific musculoskeletal soft tissues injuries. Variants within the TNC, COL5A1 and MMP3 genes co-segregate with chronic Achilles tendinopathy. The variant within the TNC gene also appears to co-segregate with Achilles tendon ruptures, while sequence variants within the COL1A1 and COL5A1 genes have been shown to be associated with cruciate ligament ruptures and/or shoulder dislocations. We proposed that genetic risk factors will in the future be included in multifactorial models developed to understand the molecular mechanisms that cause musculoskeletal soft tissue injuries or related pathology. Clinicians could eventually use these models to develop personalised training programmes to reduce the risk of injury as well as to develop treatment and rehabilitation regimens for the injured individual.



© 2012 S. Karger AG, Basel



Br J Sports Med. 2007 April; 41(4): 241–246.
Published online 2007 January 26. doi: 10.1136/bjsm.2006.033035PMCID: PMC2658952Tendon and ligament injuries: the genetic component
Alison V September, Martin P Schwellnus, and Malcolm Collins
Alison V September, Martin P Schwellnus, UCT/MRC Research Unit for Exercise Science and Sports Medicine, Department of Human Biology, Faculty of Health Sciences, University of Cape Town, Cape Town, South AfricaMalcolm Collins, UCT/MRC Research Unit for Exercise Science and Sports Medicine, South African Medical Research Council, Cape Town, South AfricaCorrespondence to: Dr M Collins
UCT/MRC Research Unit for Exercise Science and Sports Medicine, UCT, PO Box 115, Newlands 7725, South Africa; [email protected]
Author information ► Article notes ► Copyright and License information ►
Accepted December 10, 2006.
Copyright ©2007 BMJ Publishing Group and the British Association of Sport and Exercise Medicine
This article has been cited by other articles in PMC.


Abstract.

Tendons and ligaments within the upper and lower limbs are some of the more common sites of musculoskeletal injuries during physical activity. Several extrinsic and intrinsic factors have been shown to be associated with these injuries. More recently, studies have suggested that there is also, at least in part, a genetic component to the Achilles tendon, rotator cuff and anterior cruciate ligament injuries.

Tears of the rotator cuff and ACL.

In recent years, two studies have reported the investigation of the genetic susceptibility to tears of the rotator cuff and tearing of the ACL.1,20 Both studies provided evidence relating to the genetic contribution of injuries to the rotator cuff and ACL, which were based on clinical information collected from affected individuals, their siblings and matched controls.



September 29, 2010, 12:01 am Phys Ed: Are Bad Knees in Our Genes?
By GRETCHEN REYNOLDS

Are fragile knees inherited? That intriguing question motivated a new study published earlier this month in The British Journal of Sports Medicine, during which researchers looked at one family’s propensity for shredding anterior cruciate ligaments during sports. The report was part of a much larger, ongoing study of risk factors for A.C.L. injury, led by researchers affiliated with the Cincinnati Children’s Hospital and Ohio State University, which involves thousands of young athletes

In the past year, a flurry of new studies have identified several genes that seem to be closely associated with A.C.L. tears.

The gene is known to affect the suppleness of collagen or connective tissue in tendons and ligaments like the A.C.L.

But the emerging genetics of A.C.L. injury do raise the possibility that “in the not-so-distant future we might be able to screen” the DNA of young athletes and find those at highest injury risk, Dr. Hewett said.

For now, the takeaway from the current research is simple. Look to your family. “If you have a first-generation relative, a sibling or parent, who has torn an A.C.L., you are likely to be at above-average risk,” Dr. Hewett said, and might want to investigate the availability of A.C.L. injury prevention programs in your area.




I wouldnt be so sure its all Bunk ........ The Brown twins both ACLs, The Grimes boys chronic hamstring tendons, both Menzels ACLs theres got to be more to it than coincidence looks like there might be.


No i am not a scientist but i am in the medical field.
 
bandoftigers said:
For what it's worth, there is absolutely a familial predisposition to ACL ruptures. There are also several genes that code for metalloproteinases that have been associated with an increased risk of ACL ruptures, and other factors such as the anatomy of the tibia and femur, which can potentially be familial, play a role as well.
Daniel Menzels' knee injuries seemed relatively innocuous to me, and I think concern over Troys' knees is warranted, and should be factored in when considering drafting him.

Flynn RK, et al. The familial predisposition toward tearing the anterior cruciate ligament - A case control study. American Journal of Sports Medicine. 2005


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